Aphasia Wernicke ( sensory , acoustic-gnostic , receptive , fluent aphasia , deafness in words ) - aphasia (speech impairment) in case of damage to the cortical part of the auditory analyzer, Wernicke zone .
| Receptive aphasia | |
|---|---|
 Brock and Wernicke areas | |
| ICD-10 | F 80.2 |
| ICD-9 | 315.32 |
| Mesh | D001041 |
According to the classification of A. R. Luria , the basis is a violation of phonemic hearing, distinguishing the sound composition of words. With acoustic-gnostic aphasia, there is a loss of the ability to distinguish the sound composition of a word - phonemes (consonants that are contrasted by signs of hardness, softness, sonorous deafness, as well as all vowel sounds and their emphasis). Thus, the central defect here is a violation of understanding of speech - the phenomenon of "alienation of the meaning of the word" when it is correctly repeated.
Content
- 1 Symptoms
- 2 Reasons
- 3 Concomitant diseases
- 4 See also
- 5 Links
Symptoms
When the Wernicke region is defeated, a person hears everything, but does not understand the content of words, they do not cause him to associate; his native language sounds like a foreign language to him. Due to sensory aphasia, a person cannot distinguish and distinguish speech sounds.
Speech may remain grammatically correct, but it contains a lot of allegories, neologisms ("slang aphasia"), service words (conjunctions and prepositions). Statements are verbose, but not informative, the words seem to be randomly displayed. Gestures and facial expressions do not help convey the meaning of statements. The patient does not realize that his speech is impaired, and annoyed if he is not understood.
The patient does not understand simple questions (like “what is your name?”).
The understanding of the commands addressed to the muscles of the head and trunk (“close your eyes”, “sit down”, “turn”) is preserved - this feature helps to distinguish sensory cortical aphasia from hearing loss, mental illness or simulation.
Writing is broken.
Sensory cortical aphasia can be accompanied by severe excitement and paranoid state.
Thus, sensory cortical aphasia includes both expressive disorders and impressive disorders, and therefore, strictly speaking, is purely “sensory” or “receptive”. Repetition, naming, reading and writing are also violated.
Reasons
The lesion is usually localized in the posterior parts of the speech system and at least partially captures the sensory center of speech. The most common cause is an embolism of one of the lower branches of the middle cerebral artery, in particular an embolism of the posterior temporal artery or an embolism of an angular gyrus artery.
Other causes are intracerebral hemorrhage , cerebral infarction , severe traumatic brain injury and neoplasms .
Concomitant diseases
Sensory cortical aphasia is often accompanied by right-sided full hemianopsia or upper quadrant hemianopsia, in some cases there is a slight smoothing of the right nasolabial fold - there are usually no other disorders. A speech full of neologisms and paraphases, an excited state, and the absence of neurological symptoms sometimes make one suspect a mental illness of TIR . The correct diagnosis can be made on the basis of other characteristic signs of aphasia and the absence of mental illness before its development.
See also
- Aphasia Broca
- Dysgraphia
- Logorea
- Karl Wernicke
Links
- Sensory cortical aphasia (Wernicke aphasia) . Archived July 23, 2013.
- Luria A.R. Brain lesions and cerebral localization of higher mental functions . Archived July 23, 2013.